mariandina work

Diabetes Mellitus

How Does Mariandina Work – Part 1

After the introduction of Mariandina B we were still faced with death caused by complications of AIDS-like Kaposi sarcoma, which is a cancer. We were also concerned about the prevalence of genital sores caused by a virus called herpes simplex. This virus causes very painful sores on the male and female genital organs that resist treatment with antibiotics. The fact that Kaposi sarcoma (the cancer so common nowadays) has no effective treatment anywhere in the medical world, it became our number one priority.

Twelve months of hard thinking and planning went into solving this scourge. The result was the birth of Mariandina J. This capsule has some properties we were looking for. It has a purely herbal composition derived from nature’s pharmaceutical arsenal. Its activity is still to be explored fully but our present experience is that a genital herpes responds to it very rapidly. It has no known side effects as of now, if given in the recommended dosage.

Diabetes Mellitus:

Twenty of our patients who had diabetes mellitus required no more insulin after 6 months of using Mariandina A & B. A separate report to follow.

HIV Replication:

Spread of HIV is through body secretions like semen, saliva, blood and blood products. Insect bites like those of mosquitoes (which suck blood using a tubular proboscis just like drug abusers transmit HIV through dirty needles) use dirty used proboscis from one individual to another, creating a similar mechanical spread. Residual blood in the lumen of the proboscis is discharged into the second victim together with the saliva the mosquito injects to stop blood coagulation.
We know the HIV virus is a retrovirus of the Lentiform sub-group, in which the herpes virus also belongs. It consists of an envelope derived from tissue cells and carries specific molecules on its surface, e.g. GP 120. The virus has a core of RNA genome that is transcribed into the DNA of the cell using reverse transcriptase enzyme. It has a prodigious capacity for replication and mutation. Mutation of HIV is a result of the error-prone reverse transcriptase in transcribing the over 9000 bases of the virus to DNA. When the HIV is introduced in the body, the infected individual enters a stage that usually is asymptomatic and lasts for an average of 2-10 years. It is noteworthy that the duration of the asymptomatic stage varies greatly and during this period a variety of cytokines (including interferon (IFN) and IFN-gamma ARE) produced as the immune response to the HIV infection. Most patients as a probable consequence of the interferon response, display raised serum levels of Neopterin and B2-microglobulins (2 M). Initially, most patients display lymphopenia, primarily due to low numbers of CD4 cells. Later, lymphocyte counts rise because of increased numbers of CD8 cells that peak several weeks after the onset of the primary infection. These markers include soluble CD4 receptors, soluble CD8, Neopterin IFN alpha and beta. The antibody response comprises a transient response of 1gM and 1gG antibodies peaking after 3-4 weeks.

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